Benefits of Vinegar Baths

Benefits of Vinegar Baths

Benefits of Vinegar Baths

There are many types of baths that can be used for detoxification and relaxation, but none that benefit the skin with a single ingredient as much as vinegar baths.

I’m not a fan of the smell of vinegar, but I’m a huge fan of vinegar baths because they leave my skin and hair looking and feeling amazing.

Why Vinegar Baths?

Apple Cider Vinegar is a versatile natural remedy and natural home product. It can be infused with herbs to be a natural cold remedy and health tonic, or sipped to relieve heartburn.

Sprayed on skin, it takes the sting out of sunburn and as a foot soak it helps alleviate athlete’s foot.

It is also a beneficial natural beauty ingredient in several ways:

For Its Vitamin and Mineral Content

Apple Cider Vinegar (ACV) is a natural source of B-vitamins, Vitamin C and trace minerals, making it nourishing to skin. Since the skin is the body’s largest organ, it is possible to obtain these nutrients from soaking and to nourish the skin. It is naturally nourishing and moisturizing and can soften skin and hair.

Beneficial Acids

Vinegar is naturally acidic so it helps restore balance to the skin’s pH which should be slightly acidic (here’s why). Though it helps restore the skin’s pH it has an alkalizing effect internally and can also help aid digestion for this reason.

Also, many people with joint problems notice improvement from soaking in apple cider vinegar baths, and these beneficial acids and vitamins may be part of the reason.

Soothes Skin Problems

ACV can help naturally kill fungus and bacteria on the skin and offers relief for many with eczema and other skin conditions. Vinegar is often recommended for skin problems like eczema, dandruff and dry skin. These same antibacterial properties and beneficial acids make vinegar effective against zits. A tiny dab of ACV can often help reverse a zit overnight.

For this reason, AVC baths are also sometimes recommended for urinary tract infections as the vinegar can help kill the yeast or fungus and create an environment where it is difficult for infection to thrive. ACV is also a potential remedy for warts and athlete’s foot for this reason.

Reduce Body Odor

Body odor occurs when bacteria mixes with sweat or moisture from the body and thrives in the warm moist environment, especially in places like the underarms. Vinegar can help kill this bacteria, reducing odor. It also creates an environment where odor is less likely to thrive.

I haven’t personally tried it, but many people who can’t tolerate deodorants containing baking soda claim that using diluted apple cider vinegar works wonderfully as a deodorant. I would think that an armpit detox would also be helpful for those with body odor or skin irritation in the underarms.

How to Take a Vinegar Bath

Once or twice a week, I fill a tub with warm to hot water and add 1-2 cups of apple cider vinegar.

I soak for 20-30 minutes, using a washcloth to clean my face and making sure to get my hair wet as well.

After bathing, I rinse off in a cool shower, though some sources recommend letting the vinegar water dry on the skin.

Other Skin Uses for ACV

  • As I mentioned, a tiny dab of ACV can often remedy a zit overnight
  • I also use a diluted spray of apple cider vinegar (1 tablespoon in 1 cup of water) as a soothing facial toner
  • This same spray can be used on the underarms for a natural deodorant, though some people find that they need to increase the concentration of vinegar for this use
  • Dab undiluted vinegar on toenail fungus or soak in a strong (50:50) solution of white or apple cider vinegar and water.
  • Dab ACV on warts
  • Rinse hair with 1/4 cup ACV in 1 cup water for shiny hair.

How do you use vinegar? Ever taken a vinegar bath?

Benefits of Vinegar Baths originally written by Wellness Mama.


Fisetin may slow or prevent neurodegneration and can be utilised as neuroprotective agent against Alzheimer’s and Parkinson’s disease.

PMID:  J Nutr Biochem. 2015 Aug 8. Epub 2015 Aug 8. PMID: 26411262 Abstract Title:  Dietary flavonoid fisetin regulates aluminium chloride-induced neuronal apoptosis in cortex and hippocampus of mice brain. Abstract:  Dietary flavonoids have been suggested to promote brain health by protecting brain parenchymal cells. Recently, understanding the possible mechanism underlying neuroprotective efficacy of flavonoids is of great interest. Given that fisetin exerts neuroprotection, we have examined the mechanisms underlying fisetin in regulating Aβ aggregation and neuronal apoptosis induced by aluminium chloride (AlCl3) administration in vivo. Male Swiss albino mice were induced orally with AlCl3 (200 mg/kg. b.wt./day/8 weeks). Fisetin (15 mg/Kg. b.wt. orally) was administered for 4 weeks before AlCl3-induction and administered simultaneously for 8 weeks during AlCl3-induction. We found aggregation of Amyloid beta (Aβ 40-42), elevated expressions of Apoptosis stimulating kinase (ASK-1), p-JNK (c-Jun N-terminal Kinase), p53, cytochrome c, caspases-9 and 3, with altered Bax/Bcl-2 ratio in favour of apoptosis in cortex and hippocampus of AlCl3-administered mice. Furthermore, TUNEL and fluoro-jade C staining demonstrate neurodegeneration in cortex and hippocampus. Notably, treatment with fisetin significantly (P

Bisdemethoxycurcumin attenuated gastric adenocarcinoma growth by inducing mitochondrial dysfunction.

PMID:  Oncol Lett. 2015 Jan ;9(1):270-274. Epub 2014 Nov 7. PMID: 25435973 Abstract Title:  Bisdemethoxycurcumin attenuates gastric adenocarcinoma growth by inducing mitochondrial dysfunction. Abstract:  Bisdemethoxycurcumin (BDMC) is a demethoxy derivative of curcumin. In this study, a human gastric adenocarcinoma xenograft model was generated in vivo using nude mice and BDMC was observed to suppress the growth and activity of tumors, in addition to improving the physical and mental capacity of the mice. An increased number of apoptotic cells, decreased ratio of B-cell lymphoma 2 (Bcl-2)/Bcl-2-associated X protein and increased caspase-3 expression was also observed following treatment with BDMC, indicating that BDMC may promote apoptosis in tumors via mitochondrial modulation. The growth of SGC 7901 gastric cancer cells was inhibited and arrested at G1 phase. Specific indicators of mitochondrial dysfunction, a reduction in adenosine triphosphate generation, the inner mitochondrial membrane potential, augmentation of reactive oxygen species production and cytochrome c were also detected in the mitochondria following treatment with BDMC. These results indicate that BDMC attenuates gastric adenocarcinoma growth by inducing mitochondrial dysfunction.

Oral curcumin likely reduces pain associated with DOMS with some evidence for enhanced recovery of muscle performance.

PMID:  Eur J Appl Physiol. 2015 Aug ;115(8):1769-77. Epub 2015 Mar 21. PMID: 25795285 Abstract Title:  Curcumin supplementation likely attenuates delayed onset muscle soreness (DOMS). Abstract:  INTRODUCTION: Oral curcumin decreases inflammatory cytokines and increases muscle regeneration in mice.PURPOSE: To determine effects of curcumin on muscle damage, inflammation and delayed onset muscle soreness (DOMS) in humans.METHOD: Seventeen men completed a double-blind randomized-controlled crossover trial to estimate the effects of oral curcumin supplementation (2.5 g twice daily) versus placebo on single-leg jump performance and DOMS following unaccustomed heavy eccentric exercise. Curcumin or placebo was taken 2 d before to 3 d after eccentric single-leg press exercise, separated by 14-d washout. Measurements were made at baseline, and 0, 24 and 48-h post-exercise comprising: (a) limb pain (1-10 cm visual analogue scale; VAS), (b) muscle swelling, (c) single-leg jump height, and (d) serum markers of muscle damage and inflammation. Standardized magnitude-based inference was used to define outcomes.RESULTS: At 24 and 48-h post-exercise, curcumin caused moderate-large reductions in pain during single-leg squat (VAS scale -1.4 to -1.7; 90 %CL:±1.0), gluteal stretch (-1.0 to -1.9; ±0.9), squat jump (-1.5 to -1.1; ± 1.2) and small reductions in creatine kinase activity (-22-29 %; ±21-22 %). Associated with the pain reduction was a small increase in single-leg jump performance (15 %; 90 %CL ± 12 %). Curcumin increased interleukin-6 concentrations at 0-h (31 %; ±29 %) and 48-h (32 %; ±29 %) relative to baseline, but decreased IL-6 at 24-h relative to post-exercise (-20 %; ±18 %).CONCLUSIONS: Oral curcumin likely reduces pain associated with DOMS with some evidence for enhanced recovery of muscle performance. Further study is required on mechanisms and translational effects on sport or vocational performance.

Nausea severity and the number of vomiting episodes were significantly lower in the Ginger intervention group than in the control group.

PMID:  Clin J Oncol Nurs. 2015 Oct 1 ;19(5):E92-E97. PMID: 26414587 Abstract Title:  Oral Intake of Ginger for Chemotherapy-Induced Nausea and Vomiting Among Women With Breast Cancer. Abstract:  BACKGROUND: Chemotherapy-induced nausea and vomiting (CINV) is among the most common and distressing symptoms experienced by patients receiving cancer treatment. Nurses play a substantial role in the prevention and management of CINV. Ginger (Zingiber officinale Roscoe) is often advocated as beneficial for nausea and vomiting. Whether the herb is truly efficacious for this condition is, however, still a matter of debate.OBJECTIVES: This experimental randomized, controlled trial was done to assess the effect of ginger on chemotherapy-related nausea and vomiting.METHODS: All patients in the study (N = 60) received standard antiemetic drugs. The patients in the study group (n = 30) also received oral ginger for the first three days of the chemotherapy cycle. No intervention was performed in the control group (n = 30) except for the routine antiemetic treatment. Nausea severity and the number of vomiting and retching episodes were measured four times each day for the first five days of the chemotherapy cycle in the patient diary. Nausea severity was evaluated using a numeric scale ranging from 0 (no nausea) to 10 (very severe nausea).FINDINGS: The researchers analyzed the five-day mean score of nausea severity and the number of vomiting and retching episodes. Based on this comparison, nausea severity and the number of vomiting episodes were significantly lower in the intervention group than in the control group (p>0.05). However, the change in the number of retching episodes between the intervention and control groups was not statistically significant (p>0.05).

Curcumin intake has some beneficial effects on the recovery of eccentric exercise induced muscle damage.

PMID:  Eur J Appl Physiol. 2015 Sep ;115(9):1949-57. Epub 2015 Apr 29. PMID: 25921600 Abstract Title:  Attenuation of indirect markers of eccentric exercise-induced muscle damage by curcumin. Abstract:  PURPOSE: Polyphenolic curcumin is known to have potent anti-inflammatory effects; thus the present study investigated the hypothesis that curcumin ingestion would attenuate muscle damage after eccentric exercise.METHODS: Fourteen untrained young men (24 ± 1 years) performed 50 maximal isokinetic (120°/s) eccentric contractions of the elbow flexors of one arm on an isokinetic dynamometer and the same exercise with the other arm 4 weeks later. They took 150 mg of curcumin (theracurmin) or placebo (starch) orally before and 12 h after each eccentric exercise bout in a randomised, crossover design. Maximal voluntary contraction (MVC) torque of the elbow flexors, range of motion of the elbow joint, upper-arm circumference, muscle soreness, serum creatine kinase (CK) activity, and plasma interleukin-6 (IL-6) and tumor necrosis factor-α (TNF-α) concentration were measured before, immediately after, and 24, 48, 72 and 96 h after each eccentric exercise. Changes in these variables over time were compared between curcumin and placebo conditions by two-way repeated measures ANOVA.RESULTS: MVC torque decreased smaller and recovered faster (e.g., 4 days post-exercise: -31 ± 13 % vs. -15 ± 15 %), and peak serum CK activity was smaller (peak: 7684 ± 8959 IU/L vs. 3398 ± 3562 IU/L) for curcumin than placebo condition (P

Curcumin was found to have anti-hyperalgesic activity by decreasing oxidative stress and down regulation of inflammatory cytokines.

PMID:  Neurochem Res. 2015 Mar ;40(3):463-72. Epub 2014 Dec 6. PMID: 25479948 Abstract Title:  Curcumin attenuates CFA induced thermal hyperalgesia by modulation of antioxidant enzymes and down regulation of TNF-α, IL-1β and IL-6. Abstract:  Reactive oxygen species are signaling mediators of nociceptive pathways. Exogenous administrations of antioxidants show anti-hyperalgesic effect. However, very little is known about the role of endogenous antioxidant defense system in pain pathology. Curcumin is a dietary antioxidant which shows ameliorative effect on thermal hypersensitivity, however detailed study is lacking. Present study was aimed to analyze the changes in oxidative stress, modulation of antioxidant enzymes and pro-inflammatory cytokines in complete Freund’s adjuvant induced inflammatory hyperalgesia and the effect of curcumin on antioxidant defense system and pro-inflammatory cytokines. Anti-hyperalgesic activity of curcumin was evidenced after 6 h of treatment. Oxidative stress was evidenced in paw skin and spinal cord of hyperalgesic rats by high level of lipid peroxidation. A decrease in activity of antioxidant enzymes like catalase, superoxide dismutase, glutathione peroxidase, glutathione reductase and an increase in level of pro-inflammatory cytokines TNF-α, IL-1β and IL-6 in paw skin was observed as compared to normal rats. However, activity of antioxidant enzymes was enhanced in spinal cord. The changes were brought towards normal level after curcumin treatment. The results suggest that modulation of antioxidant defense system is early event in initiation of inflammatory hyperalgesia which might lead to initiation of other signaling pathways mediated by lipid peroxide, TNF-α, IL-1β and IL-6. Decrease in oxidative stress and down regulation of these cytokines by curcumin is suggested to be involved in its anti-hyperalgesic effect.